Antisense oligonucleotide eluforsen improves CFTR function in F508del cystic fibrosis

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Tezacaftor/Ivacaftor in Subjects with Cystic Fibrosis and F508del/F508del-CFTR or F508del/G551D-CFTR.

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Decoding F508del Misfolding in Cystic Fibrosis

The functional deficiency of the cystic fibrosis transmembrane conductance regulator (CFTR), a plasma membrane chloride channel, leads to the development of cystic fibrosis. The deletion of a phenylalanine at residue 508 (F508del) is the most common cause of CFTR misfolding leading to the disease. The F508del misfolding originates in the first nucleotide-binding domain (NBD1), which induces a g...

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ژورنال

عنوان ژورنال: Journal of Cystic Fibrosis

سال: 2019

ISSN: 1569-1993

DOI: 10.1016/j.jcf.2018.10.015